Kellen Kovalovich, M.D. - Medical Doctor - Endoscopy Associates of Valley Forge | LinkedIn
The Scranton Journal Fall 1988 - University of Scranton PR Publications and Press Releases - The University of Scranton Digital Collections
Deficiency of G-protein-coupled bile acid receptor Gpbar1 (TGR5) enhances chemically induced liver carcinogenesis. - Abstract - Europe PMC
Cytokine control of developmental programs in normal hematopoiesis and leukemia | Oncogene
1987 Commencement Program, University Archives, University of Pennsylvania
Steatosis does not impair liver regeneration after partial hepatectomy | Laboratory Investigation
Bile Acid Regulation of C/EBPβ, CREB, and c-Jun Function, via the Extracellular Signal-Regulated Kinase and c-Jun NH2-Terminal Kinase Pathways, Modulates the Apoptotic Response of Hepatocytes | Molecular and Cellular Biology
Aria Kovalovich - Researcher - United States Senate Committee on the Budget | LinkedIn
Corey Kovalovich - Student - The Ohio State University | LinkedIn
Deficiency of G-protein-coupled bile acid receptor Gpbar1 (TGR5) enhances chemically induced liver carcinogenesis. - Abstract - Europe PMC
Steatosis does not impair liver regeneration after partial hepatectomy | Laboratory Investigation
Sustained Phosphorylation of Bid Is a Marker for Resistance to Fas-Induced Apoptosis During Chronic Liver Diseases
Inhibition of GSK3 differentially modulates NF-κB, CREB, AP-1 and β-catenin signaling in hepatocytes, but fails to promote TNF-α-induced apoptosis - ScienceDirect
Inhibition of GSK3 differentially modulates NF-κB, CREB, AP-1 and β-catenin signaling in hepatocytes, but fails to promote TNF-α-induced apoptosis - ScienceDirect
Inhibition of GSK3 differentially modulates NF-κB, CREB, AP-1 and β-catenin signaling in hepatocytes, but fails to promote TNF-α-induced apoptosis - ScienceDirect
PMA MEDICAL SPECIALISTS - GASTROENTEROLOGY CENTER - 13 Reviews - 420 W Linfield-Trappe Rd, Limerick, PA - Yelp
Inhibition of GSK3 differentially modulates NF-κB, CREB, AP-1 and β-catenin signaling in hepatocytes, but fails to promote TNF-α-induced apoptosis - ScienceDirect
Inhibition of GSK3 differentially modulates NF-κB, CREB, AP-1 and β-catenin signaling in hepatocytes, but fails to promote TNF-α-induced apoptosis - ScienceDirect
Deficiency of G-protein-coupled bile acid receptor Gpbar1 (TGR5) enhances chemically induced liver carcinogenesis. - Abstract - Europe PMC
Braden Kovalovich - Drexel University - Philadelphia, Pennsylvania, United States | LinkedIn
Steatosis does not impair liver regeneration after partial hepatectomy | Laboratory Investigation
Liver Regeneration Edited by Dieter Häussinger
Steatosis does not impair liver regeneration after partial hepatectomy | Laboratory Investigation
